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. 2020 Oct 15;39:218. doi: 10.1186/s13046-020-01730-8

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — SETDB1 was upregulated in glioblastoma tissues and corelative with poor tumor progression. a Representative IHC staining of SETDB1 protein expression in glioblastoma tumor tissues (T) and adjacent normal tissue (N) of three patients. Scale bar: 50 μm. b mRNA level of SETDB1 was analyzed by real-time PCR in glioblastoma tumor tissues (T) and adjacent normal tissue (N) of patients. c Western blotting of SETDB1 expression in NHAs and indicated glioblastoma cells. d mRNA level of SETDB1 was analyzed by real-time PCR in NHAs and indicated glioblastoma cells. e Kaplan-Meier survival analysis indicated that glioblastoma patients with high expression of SETDB1 had worse relapse-free survival