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. 2020 Aug 25;29:0963689720946277. doi: 10.1177/0963689720946277

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© The Author(s) 2020

This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (https://creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 2. — EPC numbers and alterations. EPCs from atherogenic diet-fed animals suffered apoptosis, as shown by the elevated cleaved caspase-3 levels in STZ7 and CIT7 groups (A). However, circulatory EPC counts were reduced in diabetic animal groups (STZ4 and STZ7) compared with control groups (CIT4 and CIT7) (B) as diabetic EPCs show lower but not significantly modified levels of the chemotaxis-mediating CXCR4 (C) and the TRF2 levels seemed to be higher in EPCs from diabetic animals, but no statistical significance was noted among groups (D) (*P < 0.05, **P < 0.01 in one-way ANOVA and ^# P < 0.05 in two-way ANOVA). ANOVA: analysis of variance; CIT: citrate buffer; CXCR4,: C-X-C chemokine receptor type 4; EPC: endothelial progenitor cell; STZ: streptozotocin.