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Schematic representation of the Toll-Like Receptor 2 (TLR2)/IL-15/NF-κB pathway in pSS SGEC. TLR2, in response to pro-inflammatory stimuli, activate the NF-κB pathway; in the nucleus, the active NF-κB promote IL-15 gene transcription; thus, incrementing inflammatory disorders in SGs of pSS patients.
