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. 2020 Oct 13;11(41):3712–3722. doi: 10.18632/oncotarget.27759

Figure 5. Energy metabolism in the myocardium of CT26-inoculated BALB/c mice.

Figure 5

(A) Protein levels of HMGB1 and TNFα in the ascites of CT26-inoculated BALB/c mice or H9c2 cultured medium (CM). (B) Cell proliferation of H9c2 cells treated with the ascites of CT26-inoculated BALB/c mice (Ascites) or H9c2 cultured medium (Control). (C) Mitochondrial stress test (Seahorse assay) of H9c2 cells. (D) Basal OCR and spare OCR. (E) Proton leak and ATP production. (F) Oxidative stress examined by DHR. (G) Glycolytic stress test of H9c2 cells. (H) Matrix analysis of OCR and ECAR. Error bar, standard error from 3 mice. Significant differences were calculated using Student t-test. HMGB, high-mobility group box; TNF, tumor necrosis factor; OCR, oxygen consumption rate; ECAR, extracellular acidification rate; DHR, dihydrorhodamine 123.