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. 2020 Aug 20;11(1):927–940. doi: 10.1080/21505594.2020.1792080

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© 2020 The Author(s). Published by Informa UK Limited, trading as Taylor & Francis Group.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 2. — Phenotypic analysis of the 05ZYH33 and ΔhtpsA mutant strains. (a) Cell density was measured spectrophotometrically at a wavelength of 600 nm. (b) Antiserum aggregation reaction of the ΔhtpsA and 05ZYH33. (c) Observation of capsular morphology of the ΔhtpsA, ΔhtpsC, Δcps2B and the wild-type strains by transmission electron microscopy. (d) Determination and analysis of capsule thickness of the ΔhtpsA, ΔhtpsC, Δcps2B and wild-type strains. ** represents significant differences (P < 0.01), NS represents no significant differences.