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. 2020 Oct 16;11(10):871. doi: 10.1038/s41419-020-03080-x

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 7 — A The potential target genes predicted by three databases and the process of selecting candidate target. B The inverse correlation between miR-18b-5p and HIF1A. C The predicted binding sites of miR-18b-5p and HIF1A. D The relative luciferase activity after transfection of miR-NC or miR-18b-5p mimic in GBC-SD and SGC-996 cells. E The efficiency of si-HIF1A in GBC-SD cells examined by qRT-PCR. F The effect of HIF1A on GBC-SD cells proliferation determined by CCK8 assays. G The cloning ability of GBC-SD cells transfected with si-HIF1A. H The effect of HIF1A on cell cycle of GBC-SD cells. I The effect of HIF1A on the expression of CDK4 and CyclinD1 in GBC-SD cells examined by western blotting. *p < 0.05, **p < 0.01, ***p < 0.001.