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. 2020 Oct 18;11(10):876. doi: 10.1038/s41419-020-03058-9

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — a, b Both P2X7RA and P2X7RB isoforms mRNA are overexpressed in de novo AML as compared to MDS patients. a mRNA was extracted from primary leukemic cells from MDS and de novo AML patients and expression of P2X7R isoform A was compared (MDS, white, n = 10, de novo AML, black, n = 47). b mRNA was extracted from primary leukemic cells from MDS and de novo AML patients and expression of P2X7R isoform B was compared (MDS, white, n = 10, de novo AML, black, n = 47). c, d Within the AML group P2X7R isoforms are differently expressed in relapsing patients as compared to remitting patients. c Expression of P2X7R isoform A within the AML patient’s group by subdividing into de novo (black), relapsing (red), remitting (green) (de novo n = 47, relapsing n = 6, remitting n = 5). d Expression of P2X7R isoform B within the AML patient’s group by subdividing into de novo (black), relapsing (red) and remitting (green). (de novo n = 47, relapsing n = 6, remitting n = 5). Data are represented as mean ± SEM. *P ≤ 0.05; **P ≤ 0.01; ***P ≤ 0.001.