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. 2020 Oct 19;10:17618. doi: 10.1038/s41598-020-74634-4

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Epidermal anti-inflammatory cytokine levels in artificial psoriatic human and imiquimod-induced psoriasis rat skin specimens: (A) IL-10 immunolabeling of the epidermis at day 7 post-treatment in the artificial psoriatic (i–v) and rat psoriatic (vi–x) skins (magnification, × 400). (i, vi) non-treatment; (ii, vii) extraction; (iii, viii) betamethasone; (iv, ix), calcitriol (D3); and (v, x), normal skin. (xi, xii) Bar graph comparing the expression levels of epidermal IL-10 among the various groups in the artificial psoriatic (xi) and rat psoriatic (xii) skin specimens. (B) TGF-β immunolabeling of the epidermis at day 7 post-treatment in the artificial psoriatic (i–v) and rat psoriatic (vi–x) skins (magnification, × 400). (i, vi) non-treatment; (ii, vii) extraction; (iii, viii) betamethasone; (iv, ix), calcitriol (D3); and (v, x) normal skin. (xi, xii) Bar graph comparing the expression levels of epidermal TGF-β among the various groups in the artificial psoriatic (xi) and rat psoriatic (xii) skins.