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. 2020 Oct 19;11:5271. doi: 10.1038/s41467-020-19058-4

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — a Heatmaps of organoids from three patients grown and stimulated in a simultaneous experiment. Cellular apoptosis (caspase 3/7) and death (propidium iodide) were recorded over the 72-h drug treatment period to assess drug sensitivity. b End-point analysis of average organoid cellular apoptosis and death for each patient and combination treatment group. All data presented as mean values ± SEM, n = 3, and normalized to positive control. Response comparisons between the different patients revealed distinct sensitivities to specific drug regimens across both independent cell viability assays. Source data for panels a, b are available.