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. 2020 Oct 16;18(4):1559325820949071. doi: 10.1177/1559325820949071

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© The Author(s) 2020

This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (https://creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 1. — TGF-β1 promotes fibrogenic activity in human lung fibroblast via LncRNA-RP11. (A) The expression of 3 common collagenic proteins after TGF-β1 treatment. (B) Microarray and hierarchical cluster analyses. (C) The increased expression of LncRNA-RP11 after TGF-β1 treatment.Statistical significance was analyzed by 1-way ANOVA, **p < 0.01 compared to control, n = 3 in each experiment. (D) The expression of collagenic proteins in 3 different conditions. Control, LncRNA-RP11 wild type and without TGF-β1 treatment; shRNA-control, LncRNA-RP11 wild type and with TGF-β1 treatment; shRNA-LncRNA-RP11, LncRNA-RP11 knocking down and with TGF-β1 treatment.