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. 2020 Sep;8(18):1176. doi: 10.21037/atm-20-5952

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2020 Annals of Translational Medicine. All rights reserved.

Open Access Statement: This is an Open Access article distributed in accordance with the Creative Commons Attribution-NonCommercial-NoDerivs 4.0 International License (CC BY-NC-ND 4.0), which permits the non-commercial replication and distribution of the article with the strict proviso that no changes or edits are made and the original work is properly cited (including links to both the formal publication through the relevant DOI and the license). See: https://creativecommons.org/licenses/by-nc-nd/4.0.

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Copy number variation between LUADs with and without signature 4. (A) Overview of top CNVs and their variational frequency. (B) Forest graph of CNVs between patients with and without signature 4. The dots and horizontal bars denote the hazard rate and 5–95% CI. *, P<0.05; **, P<0.01; ***, P<0.001 (Fisher’s exact test). Only the genes passed a significant test (P<0.05) are listed. (C) The different prevalence of EGFR, ERBB2, CCND1, FGF3, FGF4, and FGF19 CNVs in patients with and without signature 4. LUAD, lung adenocarcinoma, CNV, copy number variation.