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. 2020 Oct 20;19:183. doi: 10.1186/s12933-020-01157-7

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — Adjusted hazard ratios (HR) and 95% confidence intervals for the secondary endpoint according to high-sensitivity C-reactive protein (hs-CRP) level quartiles in the overall study population (Panel A), in patients with diabetes mellitus (DM) (Panel B), and in those without DM (Panel C). Hazard ratios and P for trend were adjusted for left ventricular ejection fraction (≤ or > 40%), estimated glomerular filtration rate (≤ or > 60 ml/min/1.73 m²), type of acute myocardial infarction (STEMI vs. NSTEMI), and prior statin use. P for interaction between DM status and hs-CRP = 0.02