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. 2020 Jun 18;267(11):3268–3273. doi: 10.1007/s00415-020-09969-z

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — Case 1. Axial T2-weighted (a) and coronal FLAIR (b) images through the brain showing typical MS lesions in the periventricular white matter, perpendicular to the ependymal surface. Note generalised neuroparenchymal loss, with relative sparing of the temporal lobe white matter (left mesial temporal atrophy score is 2; right mesial temporal atrophy score is 1). Sagittal and coronal amyloid PET images (c) demonstrating generalised increased activity within the cortical grey matter in all lobes with complete loss of grey–white differentiation consistent with widespread amyloid deposition