Table 5.

Summarization of the antiinflammatory studies of different Terminalia species.

Terminalia sp.	Mechanism/Mode of action	Phytochemicals (compound no.)	Reference
T. coriacea (Roxb.) Wight & Arn.	Decreased paw volume and wet and dry weights of granulomatous tissue in acute and chronic models of inflammation in rats	(97) Apigenin, (36) kaempferol, (48) luteolin, (98) myricetin, (35) quercetin, and (118) rutin	(Khan et al., 2018)
T. bellerica (Gaertn.) Roxb.	Selective binding of gallic acid with the amino acids Arg120 and Glu524 from COX-2	(3) Gallic acid	(Reddy et al., 2010)
T. glaucescens Planch. ex Benth.	Inhibition of albumin denaturation and hemolysis	(99) termiglaucescin; (100) β-D-glucopyranosyl 2α, 3β, 6β-trihydroxy-23-gallylean-12-en-28-oate; (101) arjunglucoside I; (102) sericoside; (103) arjungenin; (104) sricic acid; (105) arjunetin; (106) chebuloside II; (107) 3,3’4-tri-O-methylelagic acid; (108) 3,3’-di-O-methylelagic acid; (37) β-sitosterol; and (10) stigmasterol	(Dawe et al., 2017)
T. muelleri Benth.	Reduction of paw edema in carrageenan-induced paw edema in mice. Reduced proinflammatory cytokines PGE2, TNF-α, IL-1β, and IL-6	(109) luteolin-6-C-glucoside, (110) vitexin, (4) ellagic acid, and (9) chebulinic acid	(Fahmy et al., 2017)