Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2020 Oct 22;11(10):894. doi: 10.1038/s41419-020-03029-0

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

PMC Copyright notice

Fig. 3 — a CCK-8 and colony formation assays indicated that the overexpressed MNX1-AS1 enhanced the proliferation ability of RBE cells, whereas knockdown of MNX1-AS1 elicited the opposite results in FRH0201 cells. The experiment was repeated three times for each group. (*P < 0.05, ***P < 0.001 by t-test). b Cell migration and invasion assays showed that the overexpressed MNX1-AS1 in RBE cells enhanced cell migration and invasion abilities, whereas knockdown of MNX1-AS1 elicited the opposite trends in FRH0201 cells. The experiment was repeated three times for each group. (*P < 0.05, ***P < 0.001 by t-test). c Endothelial tube formation assay demonstrated that the upregulated MNX1-AS1 enhanced cell angiogenic ability, whereas downregulation of MNX1-AS1 expression repressed cell angiogenic ability. The experiment was repeated three times for each group. (*P < 0.05, **P < 0.01 by t-test).