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Schematic pathophysiology of acute kidney injury in a critically ill patient with sepsis and nephrotoxic antibiotic treatment with selected miRNAs. AKI—acute kidney injury, GSTM1—glutathione-S-transferase Mu 1 gene, IL-6—interleukin 6, IL-1ß—interleukin 1ß, NF-κB—nuclear factor—kappa B, TNFα—tumor necrosis factor alpha, TNIP2—tumor necrosis factor alpha induced protein 3-interacting protein 2, TLR4—Toll-like receptor 4.
