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. 2020 Oct 24;11(10):911. doi: 10.1038/s41419-020-03071-y

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — a M⁶A quantitative analysis showed the percentage of m⁶A content in the CC tissue and adjacent normal tissue. b M⁶A quantitative analysis showed the percentage of m⁶A content in CC cells (SiHa, CaSki) and normal cells (HaCaT). c M⁶A quantitative analysis showed the percentage of m⁶A content in CC cells (SiHa, CaSki) transfected with METTL3 knockdown. d MeRIP-Seq analysis revealed the m⁶A peaks distributed at 3’-UTR, 5’-UTR and coding sequence (CDS). e Proportion analysis illustrated the m⁶A peaks were distributed in the 3’-UTR, CDS, 5’-UTR and stop codon. f Volcano plot of MeRIP-Seq showed the dysregulated m⁶A peaks in the CC cells. Data are displayed as mean ± standard deviation. **p < 0.01 vs. control.