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. 2020 Oct 14;20(6):361. doi: 10.3892/ol.2020.12225

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Copyright: © Gui et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 4. — Lentivirus-mediated overexpression of PLSCR1 promotes HepG2 cell invasion and adhesion in vitro. (A) Representative images of cell invasion determined by Transwell invasion assays of cells transfected with (a) shnon, (b) sh-PLSCR1, (c) Lvnon or (d) Lv-PLSCR1. (B) Quantification of the Transwell invasion assays. (C) Ln and (D) Fn adhesion assays. Data are presented as the mean ± SD. *P<0.05, **P<0.01, ***P<0.001. OD, optical density; PSCR1, phospholipid scramblase 1; sh, short hairpin RNA; lv, lentivirus; Ln, laminin; Fn, fibronectin.