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. 2020 Oct 28;18:174. doi: 10.1186/s12964-020-00663-1

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 11 — Erastin treatment inhibited tumorgenesis and development of HCC in vivo. a-c The original tumors (a), tumor volume (b) and tumor weight (c) under erastin treatment. d Histological changes of heart, liver, lung and kidneys in tumor-bearing and non-bearing mice under elastin treatment. e Weight change of mice under erastin treatment over time. f Pathological characteristics of tumor tissues and expression of Ki67 and N-cadherin in tumor tissues under erastin treatment. G Expression differences of ABCB6, FLVCR1, SLC7A11 and SLC48A1 between erastin-treated tumor tissues and vehicle-treated tumor tissues