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. 2020 Oct 29;39:229. doi: 10.1186/s13046-020-01748-y

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 8 — Upregulation of UPK1A-AS1 correlates with poor prognosis in patients with HCC. a. Expression level of UPK1A-AS1 in HCC from TCGA dataset (P < 0.001). b. The expression of UPK1A-AS1 from 50 paired HCC samples and adjacent non-tumor liver tissues from TCGA dataset (P < 0.05). c. Correlation between UPK1A-AS1 and tumor stage in patients with HCC (GEPIA). d. Kaplan-Meier analysis of overall survival in the TCGA dataset based on UPK1A-AS1 expression (P < 0.05). e-g. Kaplan-Meier analysis showed the correlation of UPK1A-AS1 and survival in indicated groups in HCC from TCGA dataset. h. Kaplan-Meier analysis of overall survival in the TCGA dataset based on UPK1A-AS1 and EZH2 expression levels (P < 0.001)