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. 2020 Sep-Oct;35(5):764–769. doi: 10.21470/1678-9741-2020-0333

Table 2.

Comparison of our study cohort with other published case series of aortic dissections and cocaine use.

Characteristics Daniel et al.[19] (2007) Dean et al.[6] (2014) Hsue et al.[20] (2002) Singh et al.[21]
(2007)
Yammine et al.[22] (2019) Our study
N 16 63 14 13 14 45
Age (years) 47±6.8 47±11 41±8,8 38±9 52.6±7.4 41±8,6
Gender (male) 12 (75%) 55 (87%) 8 (57%) 9 (69%) 10 (71.4%) 40 (88.9%)
History of nicotine abuse 16 (100%) 39 (61%) 14 (100%) 13 (100%) 11 (78.6%) 21/25 (84%)
History of hypertension 11 (69%) 52 (82%) 11 (79%) 9 (69%) 14 (100%) 29/39 (74.4%)
Time from cocaine use to symptoms (mean) 12,8 h (4-24 h) NR 12 h (0-24 h) 0-48 h NR 11 h (0-72 h)
Chest pain NR 51 (81%) NR NR NR 23/37 (62.2%)
Stanford type A 44% 33 (52.4%) 6 (43%) 4 (31%) 0 (0%) 33/44 (75%)
Stanford type B 57% 30 (47.6%) 8 (57%) 9 (69%) 14 (100%) 11/44 (25%)
Surgical treatment 50% 31 (49%) 9 (64%) - 14 (100%)* 31/39 (79.5%)
Mortality 31%
(after 1 year)
8%
(in-hospital)
- 69%
(after 1 year)
7%
(30-day mortality)
9/42 (21.4%)
(in-hospital)

NR=not reported

*

Endovascular treatment.