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. 2020 Oct 30;10:18769. doi: 10.1038/s41598-020-75107-4

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Flow cytometric dot plots of effects of GH on mitochondrial function in granulosa cells. (a, c) The MMP in GCs was lower in the PCOS-C group than in the non-PCOS control group, as shown by reduced JC-1 aggregates red fluorescence. The MMP was greater in the PCOS-T group than in the PCOS-C group (P < 0.05). (b, d) The numbers of early and late apoptotic cells were increased in the PCOS-C group compared with those in the non-PCOS and PCOS-T groups (P < 0.05). GH growth hormone, MMP mitochondrial membrane potential, PCOS polycystic ovary syndrome, PI propidium iodide, FITC fluorescein isothiocyanate. *P < 0.05 denotes statistical significance between groups.