Table 2.
Presence of further non-typing genes (either singularly or in combination) per toxinotype.
| Type | Typing Genes Combination | Total No. of Isolates per Type | Combination of Non-Typing Genes Detected | ||||
|---|---|---|---|---|---|---|---|
| None | cons-cpb2 | cpe * | cpe * + aty-cpb2 | aty-cpb2 | |||
| A | cpa-plc | 588 | 383 | 120 | NAa | NA a | 85 |
| B | cpa-plc + cpb + etx | 1 | - | - | NAa | NA a | 1 |
| C | cpa-plc + cpb | 7 | 6 | 1 | - | - | - |
| D | cpa-plc + etx | 19 | 5 | - | 9 | 3 | 2 |
| E | cpa-plc + iap | 1 | - | - | - | 1 | - |
| F | cpa-plc + cpe * | 16 | 13 (2.06%) | 1 | NA* | NA* | 2 |
| Total | 632 | 407 | 122 | 9 | 4 | 90 | |
| % of 632 | 100% | 64.40% | 19.30% | 1.42% | 0.63% | 14.24% | |
“-“ no isolates; NA = non-applicable; a gene non-compatible with the relative toxinotype;* cpe gene is included in the definition to the toxinotype F because the CPE toxin is typing in this case.