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. 2020 Oct 9;9(10):966. doi: 10.3390/antiox9100966

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© 2020 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/).

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Role of reactive oxygen species (ROS) in the mechanism of toxic effects of organophosphates on skeletal muscle. OP—organophosphate; ACh—acetylcholine; AChE—acetylcholinesterase; nAChR—nicotinic acetylcholine receptor; nNOS—neuronal nitric oxide synthases; ATP—adenosine triphosphate; SOD—superoxide dismutase; ETC—electron transport chain in mitochondria; DHPR—Dihydropyridine receptor; RyR—Ryanodine receptor; SR—Sarcoplasmic reticulum; NOX—NADPH oxidase; GPx—Glutathione peroxidase; CAT—Catalase.