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. 2020 Oct 13;10(10):1866. doi: 10.3390/ani10101866

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© 2020 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/).

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Hematoxylin–eosin staining of Huso huso. (a) Diseased animal showing the depletion of pericardial–epicardial lymphoid-like tissue compared to (b) aged-matched subject showing normal lymphoid tissue. (c) Diseased animal showing the depletion of lymphoid periarteriolar tissue and thickening of vessel wall (hyalinosis) compared to (d) aged-matched subject showing normal architecture. (e) Diseased animal showing the degeneration/atrophy of pyrenophores in the vicinity of epaxial muscles compared to (f) an aged-matched subject showing normal pyrenophores. (g) Diseased animal showing the degeneration and severe atrophy of skeletal myofibers and perimysial and endomysial edema compared to (h) an aged-matched subject showing normal skeletal muscle.

Hematoxylin–eosin staining of Huso huso. (a) Diseased animal showing the depletion of pericardial–epicardial lymphoid-like tissue compared to (b) aged-matched subject showing normal lymphoid tissue. (c) Diseased animal showing the depletion of lymphoid periarteriolar tissue and thickening of vessel wall (hyalinosis) compared to (d) aged-matched subject showing normal architecture. (e) Diseased animal showing the degeneration/atrophy of pyrenophores in the vicinity of epaxial muscles compared to (f) an aged-matched subject showing normal pyrenophores. (g) Diseased animal showing the degeneration and severe atrophy of skeletal myofibers and perimysial and endomysial edema compared to (h) an aged-matched subject showing normal skeletal muscle.