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. 2020 Nov 1;21:289. doi: 10.1186/s12931-020-01552-4

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — The comparison of sputum and blood eosinophil and neutrophil counts between patients colonised with different PPMs. Patients were categorised into four groups based on bacterial load defined by genome copies/ml of ≥ 1 × 10⁴; no colonisation (NC), colonised with Haemophilus influenzae (HI), Streptococcus pneumoniae (SP) or > 1 potentially pathogenic microorganism (PPM). Sputum neutrophil % (n = 145) (a), sputum eosinophil % (n = 145) (b), blood neutrophil counts (n = 226) (c), blood eosinophil counts (n = 226) (d), blood neutrophil percentages (n = 226) (e) and blood neutrophil–lymphocyte ratio (n = 226) (f) are shown for each group. Statistical analysis was performed using Kruskal–Wallis and Mann–Whitney U adjusted for multiple comparisons. Data represent individual patients with median. *, **Significant difference to no colonisation group (p < 0.05, < 0.01 respectively)