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. 2020 Oct 30;9(1):2381–2393. doi: 10.1080/22221751.2020.1835446

Table 2.

Virological characteristics of ten post-treatment patients who presented variants of rtL229V/M/F.

P Gen Drug usage HBV DNA change Mutation
1 B LMV 8.7b →NDn → 7.2s  rtL229V + rtL180M + rtM204V
2 B LMV 6.6b →NDn → 4.0s rtL229V + rtM204I
3* B LMV UKb →NDn → 6.1s rtL229V
4* B ETV 6.0b →4.3s → 3.1n rtL229F + rtM204I
5 C LMV 6.3b →NDn → 4.2s rtL229M + rtL180M + rtM204V
6 C ETV 6.7b →3.9s → NDn rtL229V
7 C ETV 7.6b →5.1s →NDn rtL229M
8 C LMV 7.0b →NDn → 7.1s rtL229V + rtL180M + rtM204V
9 C LdT 5.6b →NDn → 7.9s rtL229F + rtM204I
10 C LMV 5.8b →3.4n → 5.7s rtL229M

Note: P: patients; Gen: genotype; HBV DNA change: the increase or decrease in HBV DNA level(log10 IU/ml); b Baseline HBV DNA level: HBV DNA level detected prior to the therapy; n nadir HBV DNA level: the lowest HBV DNA level detected during the therapy; s HBV DNA level at the time point of sequencing; ND: HBV DNA not detected; UK: unknown HBV DNA level; LMV: lamivudine; LdT: telbivudine; ETV: entecavir; rt: reverse transcriptase; * patients with low therapy compliance.