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. 2019 Sep 30;11(11):3071–3085. doi: 10.1111/1759-7714.13142

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© 2019 The Authors. Thoracic Cancer published by China Lung Oncology Group and John Wiley & Sons Australia, Ltd

This is an open access article under the terms of the http://creativecommons.org/licenses/by/4.0/ License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited.

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HOXB7 knockdown induces cell cycle arrest in S exposure to cisplatin. Cells were treated with the indicated concentration of cisplatin (2 μg/mL for KYSE150 and 1 μg/mL for KYSE450) for 24 hours and underwent flow cytometry analysis by staining with PI/RNase stain to assess the cell cycle progression. Cell cycle was arrested in phase S for HOXB7‐knockdown cells by cisplatin treatment compared to control cells. This experiment was performed thrice in triplicates. () Control, () Cis, () HOXB7i, and () HOXB7i‐Cis.

Inline graphic — HOXB7 knockdown induces cell cycle arrest in S exposure to cisplatin. Cells were treated with the indicated concentration of cisplatin (2 μg/mL for KYSE150 and 1 μg/mL for KYSE450) for 24 hours and underwent flow cytometry analysis by staining with PI/RNase stain to assess the cell cycle progression. Cell cycle was arrested in phase S for HOXB7‐knockdown cells by cisplatin treatment compared to control cells. This experiment was performed thrice in triplicates. () Control, () Cis, () HOXB7i, and () HOXB7i‐Cis.