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. 2020 May 15;45(11):1817–1825. doi: 10.1038/s41386-020-0708-x

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — a T-maps illustrating significantly lower [¹¹C]ABP688 BP_ND in high EXT risk, high cannabis users (n = 9) compared to all other participants (n = 50) in mOFC, ventral striatum and insula with other substance use (AUDIT, smoking status and drug use other than cannabis), sex and E-isomer ratio as covariates. Cluster-level ps < 0.05, FWE corrected. b T-maps illustrating significantly lower [¹¹C]ABP688 BP_ND in high EXT risk, high cannabis users (n = 9) compared to all other participants (n = 50) in mOFC, ventral striatum and insula without other substance use as covariates (AUDIT, smoking status and drug use other than cannabis). Sex and E-isomer ratio were included as covariates. Peak-level ps < 0.05, FWE corrected.