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. Author manuscript; available in PMC: 2022 Dec 2.
Published in final edited form as: Immunobiology. 2022 Jun 2;227(4):152234. doi: 10.1016/j.imbio.2022.152234

Table 1. Clinical characteristics of the study participants.

Viremic Controllers [VCs, n = 07] Viremic Non-Progressors [VNPs, n = 12] Putative Progressors [PuPs, n = 11]
Agea (Years), Range 42 (29–60) 39 (30–49) 35 (21–59)
Gender Female = 03
Male = 04
Female = 04
Male = 08
Female = 06
Male = 05
CD4+ T cell count a
(cells/mm3), Range
900
(501–1469)
680
(501–910)
553
(514–908)
Viral Load a,b
(log10 copies/ml)
2.95
(1.73–3.04)
4.73
(4.01–5.35)
4.71
(3.58–5.98)
Duration of infectiona (Years), Range 10
(8–24)
10
(7–16)
1
(0.5 – 03)
Antiretroviral therapy (ART) status Naïve Naïve Naïve
HLA-B*27/B*57 status c HLA-B*27 (+ve = 0/06)
&
HLA-B*57 (+ve = 1/07)
HLA-B*27 (+ve = 1/09)
&
HLA-B*57 (+ve = 0/11)
HLA-B*27 (+ve = 02/10)
&
HLA-B*57
(+ve = 1/11)d
a

Data are expressed as the median (range).

b

Viral load was estimated at the time of sampling.

c

HLA-B*27 and HLA-B*57 allele status of 5 and 1 of the HIV-1 infected participants respectively, was not available due to lack of SSP-PCR amplification.

d

Only one participant was positive for both HLA-B*27 and HLA-B*57 allele.