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. 2020 Oct 22;44(6):2645–2655. doi: 10.3892/or.2020.7821

Figure 5.

Figure 5.

miR-505-3p reversed the effect of KTN1-AS1 on glioma cells. (A) miR-505-3p was upregulated following KTN1-AS1 knockdown in T98G and U251 cells. (B) Expression of miR-505-3p in T98G cells co-transfected with si-KTN1-AS1 + miR-505-3p inhibitor and with si-KTN1-AS1 + miR-inhibitor NC and in cells co-transfected with si-KTN1-AS1 + miR-505-3p inhibitor compared with si-KTN1-AS1 + miR-inhibitor NC. (C) Cell viability was conducted by CCK-8 assay. (D) Invasion ability was explored by Transwell assays. Magnification, ×100; scale bar, 50 µm. Data are presented as the mean ± standard error of at least 3 independent experiments. *P<0.05, **P<0.01 and ***P<0.001; #P<0.05, ##P<0.01 and ###P<0.001. *P<0.05 vs. NC. miR, microRNA; KTN1-AS1, Kinectin 1 Antisense RNA 1; GBM, glioblastoma; NC, negative control; si, small interfering.