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. 2020 Oct 22;44(6):2645–2655. doi: 10.3892/or.2020.7821

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Copyright: © Mu et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 6. — KTN1-AS1 knockdown inhibited tumor formation in vivo and prolonged mice survival. (A) Luminescence imaging showed that the BLI value of si-KTN1-AS1-treated T98G-Luc glioma cell group was suppressed compared with that of the si-NC group. (B) Line chart showed the change of tumor BLI value, which indicated the tumor size in mice. (C) Kaplan-Meier analysis indicated that mice inoculated with si-KTN1-AS1 exhibited a significantly better prognosis than those in the si-NC-treated group. *P<0.05. BLI, bioluminescence imaging; Luc, luciferase; KTN1-AS1, Kinectin 1 Antisense RNA 1; NC, negative control; si, small interfering.