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. 2020 Oct 22;8:585879. doi: 10.3389/fcell.2020.585879

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Copyright © 2020 Li, Chang, Li, Li, Gao, Zhou, Wu, Bai, Dong, Ma, Zhang, Lu, Tan, Wang and Lan.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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A model for mechanisms of cardiac hypertrophy in RAD deficient CMs. RAD deficiency triggers hypertrophy-associated pathway via modulation of the cytoplasmic Ca^{2 +}. NCX, sodium-calcium exchanger; LTCC, L-type calcium channel; CaM, Calmodulin; CaMKII, calmodulin-dependent kinase II; CnA, Calcineurin; SR, sarcoplasmic reticulum.