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. 2020 Nov 5;11:61. doi: 10.1186/s13293-020-00335-2

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 7 — Differentially targeted genes in astrocytes were involved in both life-sustaining and disease-relevant pathways. a Bubble plot shows significantly enriched KEGG pathways (having at least five annotated genes) with an FDR < 0.1. Gene ratio stands for the proportion of differentially targeted genes in corresponding pathways. KEGG pathways of b thermogenesis and c Huntington disease are illustrated. Differentially targeted genes are colored red (female-specific) or blue (male-specific). Saturation is commensurate to the difference in summed edge weight