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. 2020 Nov 5;21:217. doi: 10.1186/s12881-020-01149-0

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — 3T Brain MRI of the patient: a to d were performed when he was first transfer to our hospital at 4-month-old. a and b: axial T2WI showed arc-shaped subdural empyema was seen in bilateral frontotemporal parietal lobes. c: axial T2 Flair showed arc-shaped subdural empyema and bilateral cerebral hemisphere atrophy. d: Coronal T1WI enhanced scanning showed bilateral frontotemporal parietal dura enhanced which was called Mercedes-Benz sign. e to h was reexamined 7 days after hospitalization. e and f: axial T2WI showed new symmetrical hypersignal lesions in bilateral basal ganglia, thalamus and cerebral peduncle. g: the lesions had a slightly higher signal on T2Flair. h: the lesions had no obvious enhancement on T2Flair