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. 2020 Nov 7;20:180. doi: 10.1186/s12894-020-00751-x

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — Diseased HIC samples exhibited increased expression of immune response and ER stress proteins, with a decrease in cellular adhesion proteins. Association map for proteins differentially expressed in diseased Hunner’s IC biopsies compared to diseased non-Hunner’s IC biopsies. Nodes in the green box represent proteins with increased expression in diseased HIC tissue, while nodes within the red outline represent proteins with decreased expression. Red nodes represent proteins with an immune response function, blue represents ER protein processing/stress response, and yellow represent cellular adhesive proteins. Lines between nodes indicate reported associations as detailed in “Methods” section