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. 2020 Nov 6;10:19232. doi: 10.1038/s41598-020-75301-4

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Cardiac redox status in heart and effect of TLR 4 activation on myocardial (A) ROS (B) Reduced glutathione (C) Catalase activity (D) SOD enzyme activity, (E–G) Protein expression NRF-2 and MnSOD. Whole gel stain was used for loading control. (H) Mitochondrial oxidative stress in H9c2 cells after TLR4 activation. Data shown as Mean ± SEM, (N = 3 for western blot, N = 5 for enzyme activity) *p < 0.05, **p < 0.01 vs CON.