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. 2020 Oct 26;14:538404. doi: 10.3389/fnins.2020.538404

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Copyright © 2020 Rathore, Arora, Rastogi, Akhtar, Singh and Samim.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Effect of nanosilymarin on neurobehavioral deficits like rota-rod, spontaneous motor activity (SMA), flexion test (FT), and grip strength test for control (C), ischemic (I), NS1 (10 μg/kg b.wt), NS2 (100 μg/kg b.wt), NS3 (1,000 μg/kg b.wt), and free silymarin (Sil) (100 mg/kg b.wt). The neurological deficit was significant in the ischemic (I) group in comparison to the control (C) group. Treating the animals with nanosilymarin has decreased neurological deficit, especially at NS3 dose. Values are expressed as mean ± SEM. Results on comparison with the control group showed a significant difference (^###p < 0.001). Results obtained showed significant difference from ischemic group (^nsp > 0.05, ^∗p < 0.05, ^∗∗p < 0.01, and ^∗∗∗p < 0.001). The experiment was conducted for n = 6 data sets and results represented as mean ± SEM.