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. 2020 Sep 10;94(12):4159–4172. doi: 10.1007/s00204-020-02895-z

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — a Cytotoxicity and micronuclei formation in HepG2 cells (upper left), HepG2-CYP3A4 cells (upper right) and micronuclei levels (lower, bars) in HepG2-CYP3A4 cells treated with senecionine. The cytotoxicity in HepG2-CYP3A4 cells under the conditions of the micronuclei assay (with recovery) is also shown (-■-). Data represent mean ± S.D. from n = 3 independent experiments. b Cytotoxicity and micronuclei formation in HepG2 cells (upper left), HepG2-CYP3A4 cells (upper right) and micronuclei levels (lower, bars) in HepG2-CYP3A4 cells treated with lycopsamine. The cytotoxicity in HepG2-CYP3A4 cells under the conditions of the micronuclei assay (with recovery) is also shown (-■-). Data represent mean ± S.D. from n = 3 independent experiments