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. 2020 Oct 29;20(6):388. doi: 10.3892/ol.2020.12251

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Copyright: © Du et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 6. — miR-431 modulated viability, apoptosis and glycolysis of hepatocellular cancer cells via AKAP1 under hypoxia treatment. The introduction of pc-AKAP1 on the influence of miR-431 mimic-mediated expression of AKAP1 (A) mRNA and (B) protein of hypoxia-exposed SNU-387 and Huh7 cells was determined with reverse transcription-quantitative PCR and western blotting, respectively. The enhancement of AKAP1 on the impacts of miR-431 mimic-mediated (C) viability, (D) apoptosis and (E and F) glycolysis of hypoxia-disposed SNU-387 and Huh7 cells were evaluated using Cell Counting kit-8, flow cytometry and glycolysis assays. *P<0.05. miR, microRNA; AKAP1, A-kinase anchor protein 1; H, hypoxic; NC, negative control.