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The zinc finger E‐box binding homeobox 1 (ZEB1) knockdown in HCT116 cells resulted in reduced migratory and invasive capabilities. (A) Scratch wounds were made at 0 and 24 h after wounds (×200). (B) Quantitative measurement of wound gaps showed a larger reduction in the cellular motility of ZEB1 siRNA‐transfected HCT116 cells than control cells. (C,D) There were much fewer ZEB1 knockdown HCT116 cells that invaded through Matrigel compared to the control cells (×200).
