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. 2020 Nov 12;17:338. doi: 10.1186/s12974-020-01979-y

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — Daily levels of LCP initiators. Daily levels of ficolin-1, ficolin-2, ficolin-3, and MBL in CSF (left) and plasma (right) in patients with SAH (black) and neurologically healthy patients (green). The daily levels are presented as medians in CSF and as means in plasma. Error bars show ±1 SD. Asterisks (*) indicate significance on specific days after SAH compared to control patients (p < 0.05) using Wilcoxon’s rank-sum test. Each LCP initiator graph is based on a total number of 399 CSF samples from 63 SAH patients (mean number of samples per patient, 7; range, 6–8), 358 plasma samples from 50 SAH patients (mean number of samples per patient, 7; range, 6–8), and 12 control samples from 12 neurologically healthy patients. MBL: Mannose-binding lectin, CSF: Cerebrospinal fluid, SAH: Subarachnoid hemorrhage