Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2020 Nov 12;26:107. doi: 10.1186/s10020-020-00233-8

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

PMC Copyright notice

Fig. 4 — EA treatment restored up-regulated rno-miR-183-5p and rno-miR-210-3p, and down-regulated SIN3A mRNA and protein in SIA rats (a–c Real-time PCR; d Western blot; e IHC analysis; Technical repeats n = 3; Animal number in each group N = 6; *p value < 0.05, vs sham group; **p value < 0.05, vs. SIA group). a Rno-miR-183-5p expression was the lowest in SIA rats, while EA treatment partly restored the rno-miR-183-5p expression in SIA rats. b Rno-miR-210-3p expression was the lowest in SIA rats, while EA treatment partly restored the rno-miR-210-3p expression in SIA rats. c The level of SIN3A mRNA was the highest in SIA rats, while EA treatment partly restored the SIN3A mRNA expression in SIA rats. d The level of SIN3A protein was the highest in SIA rats, while EA treatment partly restored the SIN3A protein level in SIA rats. e EA treatment reduced SIN3A expression in the hippocampus of SIA rats