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. 2020 Nov 12;3:666. doi: 10.1038/s42003-020-01386-2

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — a Western blot analysis of endogenous β-Catenin following the knockdown of WNK1 and/or WNK4 in DLD1 cells. b–d Western blot analysis of endogenous β-Catenin following the knockdown of both WNK1 and WNK4 (b), treatment with the WNK inhibitor 26016 (c) or #13 (d), or the expression of RMND5A ΔR and/or MAEA ΔR in DLD1 colorectal cancer cells.