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. 2020 Nov 3;10:590682. doi: 10.3389/fcimb.2020.590682

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Copyright © 2020 Anil and Banerjee.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Major interactions facilitating SPN adhesion to BMECs. Major interactions between SPN ligands and host cell receptors that enable adhesion of blood-borne SPN to BMECs are depicted. These involve but are not limited to phosphoryl choline–platelet activating factor receptor, PspC (CbpA)–laminin receptor, PspC–human polymeric immunoglobulin receptor, RrgA (component of pneumococcal pili)–human polymeric immunoglobulin receptor, RrgA–platelet endothelial cell adhesion molecule 1, and enolase–plasminogen interactions.