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. 2020 Nov 4;11:586729. doi: 10.3389/fmicb.2020.586729

TABLE 1.

The effect of NagZ on resistance in EC.

Strain MICs (μg/ml)
TZP ATM PIP CRO CAZ SCF
S1+pBAD33 2 1 2 0.125 0.25 0.5
S1+pBAD33-nagZ 8 128 16 128 256 8
S2+pBAD33 2 4 4 0.5 0.125 0.5
S2+pBAD33-nagZ 32 >512 64 256 512 2
S3+pBAD33 2 2 8 0.25 1 0.5
S3+pBAD33-nagZ 16 128 128 64 256 4
S4+pBAD33 4 1 8 0.5 0.5 0.5
S4+pBAD33-nagZ 32 64 64 64 256 8
S5+pBAD33 1 0.5 1 0.5 2 1
S5+pBAD33-nagZ 32 64 32 32 512 8
S6+pBAD33 0.25 1 0.5 1 0.25 1
S6+pBAD33-nagZ 16 256 32 128 64 8
R1 2 64 2 128 128 4
R-ΔnagZ 0.5 0.125 0.5 0.125 0.25 0.25
R1-ΔnagZ+pBAD33 0.5 0.125 0.5 0.25 0.5 0.25
R1-ΔnagZ+pBAD33-nagZ 8 64 8 64 64 8

EC, Enterobacter cloacae; MIC, minimum inhibitory concentration; PIP, piperacillin; TZP, piperacillin-tazobactam; ATM, aztreonam; CRO, ceftriaxone; CAZ, ceftazidime; SCF, cefoperazone-sulbactam; S, susceptible EC isolated from the clinical sample; S1, susceptible EC isolates number 1, and so on; R1, resistant Enterobacter cloacae clinical isolates number 1; R1-ΔnagZ, nagZ knockout R1; pBAD33, control vector; pBAD33-nagZ, NagZ complementation vector; R1-ΔnagZ+pBAD33, R1-ΔnagZ complemented with pBAD33; R1-ΔnagZ+pBAD33-nagZ, R1-ΔnagZ complemented with pBAD33-nagZ.