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. 2020 Nov 20;18:171. doi: 10.1186/s12951-020-00733-z

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 6 — TSC-Exos exert anti-inflammatory effects in Dox-induced cardiac injury. a ELISA analysis of the concentration of inflammatory factors, including IL-1β and IL-6; b the expression of p65 by western blotting. Dox+Vector indicates Dox-treated mice injected with AAV9 vector; Dox+AAV indicates Dox-treated mice injected with AAV9-miR-200b inhibitor; Dox+Exo indicates Dox-treated mice injected with TSC-Exos. #p < 0.05 compared with the Dox+Vector group