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. 2020 Nov 2;21(1):4. doi: 10.3892/etm.2020.9436

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Copyright: © Zhao et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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PSD95 interaction with NR2B and nNOS following Dex treatment. (A) Dex treatment reduced PSD95 interaction with NR2B and nNOS as presented by immunoprecipitation assay. (B) Relative levels of nNOS and NR2B immunoprecipitated with PSD95 and normalized to the Veh group. Data were represented as the means ± standard error of the mean. ^*P<0.05 vs. Veh. Dex, dexmedetomidine; IgG, immunoglobulin G; kD, kilodalton; nNOS, neuronal nitric oxide synthase; NR2B, N-methyl-D-aspartic acid receptor subunit; PSD95, post-synaptic density 95; TBI, traumatic brain injury; Veh, vehicle.