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. 2020 Nov 19;10:20149. doi: 10.1038/s41598-020-76864-y

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Scheme illustrating pathology of psoriasis-like lesions. Neutrophils cause NETosis, and cytokines, including IL-1β and IL-36γ are secreted by macrophages activated by NETs, and the production of IL-17A from γδ T cells is promoted by IL-1β or CCL20 from imiquimod (IMQ)-induced psoriasis-like lesions. KC: keratinocytes DC: dendritic cells MP: macrophages NETs: neutrophil extracelluar traps Neu: neutrophils. αβT: αβT cells γδT: γδT cells.