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. 2020 Nov 20;13:135. doi: 10.1186/s13048-020-00741-5

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 6 — STAT4 suppresses the effects of KISS1 on cell apoptosis and synthesis of E2 in GCs. a STAT4 repressed the anti-apoptotic effect of KISS1 in ovarian GCs; b STAT4 suppressed the synthetic capacity of E2 of KISS1 in ovarian GCs; c Changes of STAT4 mRNA expression during follicular maturation. ** indicates P < 0.01; * indicates P < 0.05. Data were represented as means ± SD